Renae Gow
Research Assistant
| telephone | +61 3 9288 2577 |
| facsmilie | +61 3 9288 2581 |
| renaeg @ medstv.unimelb.edu.au |
Profile
Ms Renae Gow works in the diabetes complications group which looks at kidney and heart disease examining the structural and functional changes that occur with disease. In particular, her role investigates gene expression studies looking at various growth factors and markers of diseased tissue which are all performed by real time pcr, and has responsibility for teaching undergraduate and postgraduates real time pcr. Renae also performs albumin radioimmunoassays, immunohistochemistry and elisa experiments on various cytokines, growth factors and proteins.
University of Melbourne find an expert profile
Professional Activities:
Member of ANZSN (Society of Nephrology)
Qualifications
BSc (Hons)
Selected Publications
- Advani A, Kelly D, Advani SL, Cox A, Thai K, Zhang Y, White KE, Gow R, Marshall SM, Steer BM, Marsden PA, Rakoczy PE & Gilbert R. 2007. Role of VEGF in maintaining renal structure and function under normotensive and hypertensive conditions. Proceedings of the National Academy of Sciences of the United States of America. 104 (36): 14448-14453.
- Langham R, Kelly D, Gow R, Zhang Y, Cordonnier DJ, Pinel N, Zaoui P & Gilbert R. 2006. Transforming growth factor-ß in human diabetic nephropathy: effects of ACE inhibition. Diabetes Care. 29 (12): 2670-5.
- Boyle A, Kelly DJ, Zhang Y, Cox AJ, Gow R, Way KJ, Itescu S, Gilbert RE & Krum H. 2005. Inhibition of protein kinase C reduces left ventricular fibrosis and dysfunction following myocardial infarction. Journal of Molecular and Cellular Cardiology. 39 ((2)): 213-221.
- Kelly DJ, Gow R, Zhang Y, Gilbert RE & Chanty A. 2005. Protein Kinase CB Inhibition Attenuates Osteopontin Expression, Macrophage Recruitment, and Tubulointerstitial Injury in Advanced Experimental Diabetic Nephropathy. Journal of the American Society of Nephrology. 16: 1654-1660.
- Kelly DJ, Zhang Y, Gow R, Itescu S & Gilbert RE. 2005. Cells expressing the stem cell factor receptor, c-kit, contribute to neoangiogenesis in diabetes. Diabetes and Vascular Disease Research. 2 (2): 76-80.
- Edgtton KL, Gow R, Kelly DJ, Carmeliet P & Kitching AR. 2004. Plasmin is not protective in experimental renal interstitial fibrosis. Kidney International. 66: 68-76.
- Kelly DJ, Zhang Y, Gow R & Gilbert RE. 2004. Tranilast attenuates structural and functional aspects of renal injury in the remnant kidney model. Journal of the American Society of Nephrology. 15: 2619-2629.
- Langham R, Kelly DJ, Gow R, Zhang Y, Dowling JK, Thomson NM & Gilbert RE. 2004. Increased expression of urotensin II and urotensin II receptor in human diabetic nephropathy. American Journal of Kidney Diseases. 44 (5): 826-831.
- Kelly DJ, Zhang Y, Hepper CL, Gow R, Jaworski K, Kemp BE, Wilkinson-Berka JLA & Gilbert RE. 2003. Protein kinase C ß inhibition attenuates the progression of experimental diabetic nephropathy in the presence of continued hypertension. Diabetes. 52: 512-518.